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The mechanisms are still to be elucidated but a likely factor is placental ischemia/hypoxia [43], but it is thought that ischemia-reperfusion injury may contribute to the oxidative stress and could result in the release of reactive oxygen species into the maternal circulation possibly resulting in oxidative DNA damage and may underlie development of fetal growth restriction [44]

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BPC-157 has demonstrated excellent tolerability across numerous preclinical studies with no significant adverse effects even at high doses